By Eberhard Nieschlag (auth.), Eberhard Nieschlag, Hermann M. Behre, Susan Nieschlag (eds.)
This is the 3rd, absolutely revised variation of a well known textbook that maintains to symbolize the highest quality within the literature on scientific andrology. It examines intensive all points of male reproductive overall healthiness, encompassing the fundamental body structure of male reproductive functionality and quite a lot of problems. all the chapters is written by way of remarkable specialists within the box. one of the issues lined are:
- Testicular function
- Sperm maturation and fertilization
- Diagnostic tactics
- Primary and secondary hypogonadism
- Late-onset hypogonadism
- Erectile dysfunction
- Genetic disorders
- Environmental influences
- Psychology and sexual medicine
- Testosterone therapy
- Assisted replica: TESE and ICSI
- Male contraception
- Ethics in andrology
This quantity will function a textbook for the newcomer to andrology, whereas supplying the skilled health professional with a helpful reference paintings. it is going to attract all who're fascinated with male reproductive overall healthiness:
- Basic scientists
Read Online or Download Andrology: Male Reproductive Health and Dysfunction PDF
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Additional resources for Andrology: Male Reproductive Health and Dysfunction
In contrast, the Ap spermatogonia divide, renew themselves and differentiate into two B spermatogonia. Detailed studies in non-human primates led to a revised model for spermatogonial expansion in men (Ehmcke and Schlatt 2006): only Ap spermatogonia divide and give rise to Ap spermatogonia (to replenish this cell pool) as well as to B-type spermatogonia for further development. In contrast to the earlier model of Clermont, Ad spermatogonia are not the source of Ap spermatogonia during regular spermatogenic cycles.
Release of sperm from the germinal epithelium into the tubular lumen (spermiation). Physiology of Testicular Function Fig. 3 Schematic representation of all germ cell types that occur in the human seminiferous epithelium. Ap spermatogonia enter the spermatogenic process (arrow on the cell indicates direction of germ cell development). Ad spermatogonia are believed to constitute the testicular stem cells. Ad = A-dark spermatogonium, Ap = A-pale spermatogonium, B = B spermatogonium, Pl = preleptotene spermatocytes, L = leptotene spermatocytes, EP = early pachytene spermatocytes, MP = mid pachytene spermatocytes, LP = late pachytene spermatocytes, II = 2nd meiotic division, RB = residual body, Sa1 – Sd2 = developmental stages of spermatid maturation Spermatogoniogenesis Ad Ap B Spermatids Sd2 RB Sb1 Sb2 Spermiogenesis Ap Ad SC 1 SC 2 MP Sa1/2 Ad SC 2 Z LP II B SC 1 L PL EP Ap B SC 1 17 Meiosis 2 SC1 SC 2 SC 2 RS RS RS RS ES ES ES ES Fig.
Progesterone inhibits gonadotropin release at least in part via arcuate nucleus dopaminergic and NPY neurons (Dufourny et al. 2005). The negative feedback action of androgens and progestins is most important for the development of a male fertility control regimen (Chap. 29). Physiological pattern Nonphysiological pattern GnRH or continuous Gonadotropin secretion TESTIS GnRH analogues (receptor blockade) LH Gonadotropic cells LH Fig. 10 Importance of the pulsatile pattern of GnRH secretion for gonadotropin secretion and testicular function.